Von Brevern M, Bertholon P, Brandt T, Fife T, Imai T, Nuti D, Newman-Toker D (2015) Benign paroxysmal positional vertigo: diagnostic criteria. Kao WT, Parnes LS, Chole RA (2016) Otoconia and otolithic membrane fragments within the posterior semicircular canal in benign paroxysmal positional vertigo. Hain TC, Squires TM, Stone HA (2005) Clinical implications of a mathematical model of benign paroxysmal positional vertigo. Ĭohen HS, Sangi-Haghpeykar H (2010) Nystagmus parameters and subtypes of benign paroxysmal positional vertigo. Talaat HS, Abuhadied G, Talaat AS, Abdelaal MS (2015) Low bone mineral density and vitamin D deficiency in patients with benign positional paroxysmal vertigo. Kahraman SS, Ozcan O, Arli C, Ustun I, Erduran R, Akoglu E, Gokce C (2016) Calcium homeostasis during attack and remission in patients with idiopathic benign paroxysmal positional vertigo. Schuknecht HF, Ruby RR (1973) Cupulolithiasis. Moriarty B, Rutka J, Hawke M (1992) The incidence and distribution of cupular deposits in the labyrinth. Kao WT, Parnes LS, Chole RA (2017) Otoconia and otolithic membrane fragments within the posterior semicircular canal in benign paroxysmal positional vertigo. Roberts RA, Abrams H, Sembach MK, Lister JJ, Gans RE, Chisolm TH (2009) Utility measures of health-related quality of life in patients treated for benign paroxysmal positional vertigo. Obermann M, Bock E, Sabev N, Lehmann N, Weber R, Gerwig M, Frings M, Arweiler-Harbeck D, Lang S, Diener HC (2015) Long-term outcome of vertigo and dizziness associated disorders following treatment in specialized tertiary care: the Dizziness and Vertigo Registry (DiVeR) Study. Lopez-Escamez JA, Gamiz MJ, Fernandez-Perez A, Gomez-Finana M (2005) Long-term outcome and health-related quality of life in benign paroxysmal positional vertigo. ĭ'Silva LJ, Whitney SL, Santos M, Dai H, Kluding PM (2017) The impact of diabetes on mobility, balance, and recovery after repositioning maneuvers in individuals with benign paroxysmal positional vertigo. Neuhauser HK (2016) The epidemiology of dizziness and vertigo. Patient-perceived disability is not related to positional nystagmus intensity. This subtype might therefore require closer follow-up. This study suggests that patients with lateral canal BPPV have increased patient-perceived disability, lower vitamin D-levels and longer duration of symptoms. There was no correlation between DHI scores and nystagmus intensity. Lateral canal BPPV was associated with longer symptom duration and lower 25-hydroxyvitamin D-levels. Higher DHI scores were associated with lateral canal BPPV and female gender. Resultsġ32 patients aged 27–90 (mean 57, SD 13) years were included. Factors: age, gender, positional nystagmus intensity (maximum slow-phase velocity), symptom duration, 25-hydroxyvitamin D-level and traumatic aetiology. Outcomesĭizziness handicap inventory (DHI), posterior vs. Patients were screened for other neurological and otological disorders. This prospective observational multicentre study analysed consecutive patients with BPPV, confirmed by standardized procedures including videonystagmography under diagnostic manoeuvres in a biaxial rotational chair. Our aim was to assess dizziness handicap and clinical characteristics of posterior and lateral canal BPPV. Such differences may have clinical and therapeutic consequences. Whether these subtypes entail any significant differences in patient-reported symptoms is yet not known. Benign paroxysmal positional vertigo (BPPV) is diagnosed and divided into subtypes based on positioning vertigo and nystagmus.
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